Melody Duvall, MD, PhD
Associate in Critical Care Medicine, Division of Critical Care Medicine
Assistant Professor of Anaesthesia, Harvard Medical School
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Melody Duvall, MD, PhD
Associate in Critical Care Medicine, Division of Critical Care Medicine
Assistant Professor of Anaesthesia, Harvard Medical School
Medical Services
Languages
English
Education
Graduate School
Immunology
University of Oxford
2006
Oxford
United Kingdom
Medical School
Washington University School of Medicine
2008
St. Louis
MO
Residency
Pediatrics
Boston Combined Residency Program (BCRP)
2011
Boston
MA
Fellowship
Critical Care
Boston Children's Hospital
2015
Boston
MA
Certifications
American Board of Pediatrics (General)
American Board of Pediatrics (Critical Care Medicine)
Publications
Select Airway Specialized Pro-Resolving Mediators Are Associated with Recovery from Non-Viral COPD Exacerbations. View Abstract
An altered natural killer cell immunophenotype characterizes clinically severe pediatric RSV infection. View Abstract
15-epi-lipoxin A5 promotes neutrophil exit from exudates for clearance by splenic macrophages. View Abstract
Pediatric Respiratory Syncytial Virus Hospitalizations and Respiratory Support After the COVID-19 Pandemic. View Abstract
Vitamin D constrains inflammation by modulating the expression of key genes on Chr17q12-21.1. View Abstract
Resolvin D2 regulates type 2 inflammatory responses and promotes resolution of mouse allergic inflammation. View Abstract
The Maresin 1-LGR6 axis decreases respiratory syncytial virus-induced lung inflammation. View Abstract
The Impact of Insulin Resistance on Loss of Lung Function and Response to Treatment in Asthma. View Abstract
Mouse phospholipid phosphatase 6 regulates dendritic cell cholesterol, macropinocytosis, and allergen sensitization. View Abstract
Case 2-2022: An Adolescent Male in Cardiac Arrest 3 Days After Liver Transplantation for End-Stage Liver Disease. View Abstract
Specialized pro-resolving mediators in respiratory diseases. View Abstract
Inflammation resolution circuits are uncoupled in acute sepsis and correlate with clinical severity. View Abstract
Invasive and noninvasive ventilation strategies for acute respiratory failure in children with coronavirus disease 2019. View Abstract
Human NK Cell Cytoskeletal Dynamics and Cytotoxicity Are Regulated by LIM Kinase. View Abstract
Leukocyte function assessed via serial microlitre sampling of peripheral blood from sepsis patients correlates with disease severity. View Abstract
Non-type 2 inflammation in severe asthma is propelled by neutrophil cytoplasts and maintained by defective resolution. View Abstract
Neutrophil cytoplasts induce TH17 differentiation and skew inflammation toward neutrophilia in severe asthma. View Abstract
ALX receptor ligands define a biochemical endotype for severe asthma. View Abstract
Better Late Than Never? Deferred Consent for Minimal Risk Research in the ICU. View Abstract
ALX receptor ligands define a biochemical endotype for severe asthma. View Abstract
Bronchoprotective mechanisms for specialized pro-resolving mediators in the resolution of lung inflammation. View Abstract
Natural killer cell-mediated inflammation resolution is disabled in severe asthma. View Abstract
Mast cell-derived neurotrophin 4 mediates allergen-induced airway hyperinnervation in early life. View Abstract
DHA- and EPA-derived resolvins, protectins, and maresins in airway inflammation. View Abstract
Pulmonary hypertension associated with scurvy and vitamin deficiencies in an autistic child. View Abstract
Polyfunctional T cell responses are a hallmark of HIV-2 infection. View Abstract
Effects of cryopreservation on CD4+ CD25+ T cells of HIV-1 infected individuals. View Abstract
Dendritic cells are less susceptible to human immunodeficiency virus type 2 (HIV-2) infection than to HIV-1 infection. View Abstract
Maintenance of HIV-specific CD4+ T cell help distinguishes HIV-2 from HIV-1 infection. View Abstract
Analysis of human immunodeficiency virus cytopathicity by using a new method for quantitating viral dynamics in cell culture. View Abstract
Cytopathic killing of peripheral blood CD4(+) T lymphocytes by human immunodeficiency virus type 1 appears necrotic rather than apoptotic and does not require env. View Abstract